Curiously, a recent study has also evaluated the role of CXCR3 in the trafficking of NK cells to the DLN during influenza infection and found that despite the expression of CXCR3 predominantly on the immature CD27+CD11b− subset, the accumulation of CXCR3−/− NK cells in the DLN was largely unaffected (31). The gene discussed is CXCR3; the disease is influenza.