NRAS and gastric cancer: Additionally, NEAT1 modulates the expression of well-known oncogenes, such as STAT3 (signal transducer and activator of transcription 3) and NRAS (NRAS proto-oncogene, GTPase), by competing for miR-506 and let-7e in gastric cancer and glioma, respectively, with a consequent increase in growth, invasion and migration [87,88].