Another finding suggested that the CYP ω-hydroxylase, consisting of CYP4A and CYP4F molecules, promoted angiogenesis and metastatic potential in human NSCLC cells through up-regulation of Vascular endothelial growth factor (VEGF) and Matrix metallopeptidase 9 (MMP-9) (Wang et al., 2010; Bell and Strobel, 2012). This evidence concerns the gene CYP4F3 and non-small cell lung carcinoma.