Furthermore, in a mouse model of metabolic syndrome (MetS), it was shown that MetS mice displayed accentuated postprandial hypertriglyceridemia after 3 h due to a defective clearance of triglyceride-rich lipoproteins and that this was associated in a delay in the induction of MTTP, LFABP, and APOC2 linked to blunted lipid sensing by CD36 in that model. This evidence concerns the gene CD36 and metabolic syndrome.