In order to examine the therapeutic potential of hiPSC-derived renal progenitors isolated with the combination of cell surface markers identified in this study, we transplanted aggregates of hiPSC-derived CD9−CD140a+CD140b+CD271+ cells isolated on day 28 into kidney subcapsules of AKI mouse models induced with ischemia reperfusion (I/R) injury just after the injury. This evidence concerns the gene NGFR and acute kidney injury.