The same analytic strategy was used in Study 2 (e.g., Pearson correlations, PROCESS mediation analyses) [73] [74] [75] to determine whether the three rumination subtypes differentially accounted for relations between social identification (i.e., centrality, ingroup affect, ingroup ties) and mental health (i.e., symptoms of depression) or circulating cytokines (TNF-α and IL-10). The gene discussed is IL10; the disease is depressive symptom measurement.