Very similar results were achieved by using DIDS analogs (H2DIDS, SITS, DPC) which successfully counteracted cisplatin- or selenite-induced apoptosis in SH-SY5Y cells (a cell line commonly used as model of neurodegenerative diseases), again by diminishing the oligomerization of VDAC1 (Ben-Hail and Shoshan-Barmatz, 2016). This evidence concerns the gene VDAC1 and neurodegenerative disease.