The same findings were reported in dystrophic mdx mouse muscles (these spontaneous mdx mutant mice do not express dystrophin and may be useful for studying Duchenne muscular dystrophy, they are also known as DMD), which revealed that expression levels of miR-206 were widely enhanced in the muscle when correlated with normal mouse muscles [102], indeed, miR-206 levels are elevated in the diaphragm muscle of an animal mouse model of muscular dystrophy [54]. This evidence concerns the gene DMD and Duchenne muscular dystrophy.