The high concentration of TNF-α cytokines and the intensification of phagocytosis observed in our previous study in chickens receiving a zinc glycine chelate, in conjunction with the high concentration of AGP observed in the present study, indicates that these proteins interact to limit the APR by stimulating the cytotoxicity of macrophages and heterophils, which counteract infections by pathogenic microbes. The gene discussed is ATP5MK; the disease is infection.