As CXCR3-mediated recruitment appears crucial for T cell entry into tumors and as Treg reduce CXCL9 and CXCL10 expression in tumors, we were eager to determine the source of CXCL10 in tumor tissue, and determine if CXCL10-producing cells were indeed affected by Treg depletion. Here, CXCR3 is linked to neoplasm.