In a PD module, UDCA significantly attenuated programed cell death events and protected human dopaminergic SH‐SY5Y cells through the PI3K‐Akt/PKB signaling pathways, similarly, UDCA deregulated the level of rotenone‐induced apoptosis by modulating mitochondrial dysfunction (Abdelkader, Safar, & Salem, 2016; Chun & Low, 2012). The gene discussed is AKT1; the disease is Parkinson disease.