Moreover, we demonstrated that this effects owned to the increased expression of PPARγ via enhancing the expression of slc10a2, then up-regulated the expression of PTEN and down-regulated the expression of mTOR, thus increased the expression of apoptotic genes and anti-cancer genes, and reduced the expression of anti-apoptotic genes to suppress the proliferation of NSCLC cells and promote the apoptosis of NSCLC cells [34]. This evidence concerns the gene PTEN and non-small cell lung carcinoma.