MTOR and tuberous sclerosis: Indeed, aerobic glycolysis, mevalonate synthesis, Liver Kinase B1 (LKB1), 5’ AMP-activated Protein Kinase (AMPK), salt-inducible kinases, and the Tuberous Sclerosis-mammalian Target of Rapamycin complex (TSC-mTOR) all influence YAP/TAZ activity [209,321,322,323,324,325].