Our data suggest that the expanded peripheral populations we have seen have the potential to be recruited to the inflamed liver because they express high levels of the chemokine receptor CXCR3, which facilitates the recruitment of effector and regulatory T cells to the inflamed human liver2, 30 through interactions with its ligand CXCL‐10.2, 17, 30 We detected both CXCR3pos Tregs and NK cells residing close to CXCL‐10‐expressing bile ducts in liver biopsies from patients with acute AIH. This evidence concerns the gene CXCL10 and autoimmune hepatitis.