The comparative analysis with inflammation showed an overlap of three molecules (RSAD2, PLSCR1 and EPSTI1), whereas the autoimmune disease group displayed a complete overlap with the significant molecules associated with treated MS patient blood samples, both in terms of fold-change directionality and similar fold changes apart from EIF2AK2, which displayed either an up- or down-regulation in 50% of all significant data points. This evidence concerns the gene EPSTI1 and myeloid sarcoma.