Additionally, LRRK2 and Parkin recruitment to mitochondria was impaired upon CCCP treatment in sporadic PD patient-derived fibroblasts, highlighting that the LRRK2/DRP1 and PINK1/Parkin pathways act in parallel, converge on Miro and are impaired in sporadic PD [18]. The gene discussed is DNM1L; the disease is Parkinson disease.