At 180 days post‐DSS, 72% (8/11) of Apc+/minMbd2−/− mice displayed mucinous adenocarcinoma with nuclear β‐catenin (Figure 5B), in contrast to control Apc+/minMbd2−/− mice, which remained disease‐free at the same age, as reported previously16. The gene discussed is APC; the disease is colon mucinous adenocarcinoma.