Assuming, that homing of vascular progenitor cells in sepsis involves various mediators that recruit them to activated endothelium in response to a damage-induced inflammation, we set out to determine in septic patients which adhesion molecules are expressed by CD34+/CD133+-stem cells—containing vascular progenitors [17,18]—and could therefore be involved in the CD34+/CD133+-stem cell-driven repair process. Here, PROM1 is linked to Sepsis.