RET/PTC1 is generated by the fusion of RET TK domain with the 5’ terminal region of the CCDC6/H4 gene, and is associated with the classical PTC variant [5], whereas RET/PTC3 consists in the fusion between the TK domain of RET and the RFG/NCOA4 gene [6], and is usually associated with a more aggressive phenotype, particularly with the solid variant; bigger size; and a more rapid development of the tumor [7]. Here, NCOA4 is linked to neoplasm.