IL-6, VEGF, and TGF-β1 have been proved to be commonly overexpressed in human breast cancer cases [26–28], FGF10 played the important role in type III epithelial-mesenchymal transition (EMT) on breast cancer cells and initiation of metastasis [29], and high expression of FGF17 causes tamoxifen resistance [30]. The gene discussed is FGF17; the disease is breast cancer.