ABCA1 and lung adenocarcinoma: On the other hand, the lung adenocarcinoma cells proliferation could be inhibited by the over expression of the three genes above 10, 22.Our findings that efatutazone elevated the expression of PPARγ, LXRα, and ABCA1 further proved that efatutazone participates in PPARγ/LXRα/ABCA1 pathway in HCC827‐GR and PC9‐GR cells.