SLE is a multifarious and chronic inflammatory autoimmune disorder with a wide range of clinical symptoms.4, 5 In patients with SLE, the immune system is subverted to against self‐antigens and the consequent persistent inflammatory response elicits a vicious cycle of immune‐cell activation and tissue damage.6 The deregulated production of pro‐inflammatory cytokines, such as TNF‐α and IL‐6, play a key role in immune dysfunction, mediating tissue inflammation and organ damage in SLE.7 TNF‐α levels were found to increase in patients with SLE and were associated with the disease activity of SLE. Here, TNF is linked to systemic lupus erythematosus.