Emerging evidences have revealed that EMT functions importantly in cancer metastasis via the transdifferentiation of epithelial cells into motile mesenchymal cells.4 During EMT, the hallmark is a decrease in E‐cadherin expression, accompanied by an increase in N‐cadherin or vimentin expression.4, 5 Conversely, mesenchymal‐epithelial transition indicates the reverse process. The gene discussed is VIM; the disease is cancer.