VIM and gastric cancer: To demonstrate the involvement of NF‐κB signal pathway in CRYAB‐induced gastric cancer cells EMT and invasion, helenalin (1 μmol/L for 1 hour), an NF‐κB inhibitor, was used.6 The results revealed that helenalin pretreatment enhanced shCRYAB‐mediated increase in E‐cadherin and decrease in p‐NF‐κB p65, N‐cadherin and vimentin (Figure 5C), and observably potentiated shCRYAB‐inhibited cell invasion (Figure 5E).