Cyclin G1 and CAT-1 played a crucial role in cell survival and proliferation in the carbon tetrachloride- and thioacetamide-induced liver injury models [24]; however, their role in INH-induced liver injury models has yet to be evaluated under the rationale for testing Cyclin G1 and CAT-1 expression in our INH-induced liver injury model. Here, SLC7A1 is linked to injury.