EPHA2 and pulmonary sarcoidosis: These data suggest that the NOD2 2722G > C variant was not sufficient to trigger pulmonary sarcoidosis by itself and it is the presence of other variants (IL17RA 958 T > C, EPHA2 2875G > A and KALRN 28C > T) that contribute to the pathology in patients with pulmonary sarcoidosis of family X, perhaps by enhancing the development and chronicity of pulmonary sarcoidosis in family X.