Recently, Hyltén-Cavallius et al. [9] reported that patients with LQTS due to impaired function of the hERG-encoded Kv11.1 potassium channel not only have alterations in their cardiac conduction, but also exhibit increased insulin, GLP-1 and GIP secretion with risk of hypoglycemia, as well as decreased fasting and hyperglycemia induced levels of glucagon. Here, GCG is linked to Hypoglycemia.