However, parasite-specific antibody may bolster worm killing following FcR engagement on Mφ, as we observed more profound larvicidal effects upon transfer of +14 day BmL3AAMφ generated from WT infection (where anti-parasite antibody would presumably be bound to Mφ FcR) compared with in vivo IL-4R ligation and BmL3AAMφ development within SCID mice. Here, IL4R is linked to infection.