Adoptive transfer of BmL3AAMφ rendered SCID mice resistant to B. malayi infection and was dependent on CCR3+ cell recruitment in SCID recipients because αCCR3 treatment effectively nullified the sustained eosinophilia in BmL3AAMφ SCID recipients and reversed the resistant phenotype in controlling larval establishment (Fig 6G–6I). Here, CCR3 is linked to Increased total eosinophil count.