LRRK2 and brain injury: The high kinase activity of LRRK2 is associated with defects in protein synthesis and degradation, apoptosis, inflammatory responses, and oxidative damage (Tsika and Moore, 2012; Cookson, 2015; Islam and Moore, 2017), which are also factors that trigger acute brain injury following TBI (Devyatov et al., 2017; Dixon, 2017).