The proinflammatory role of IL-32 has been well reported in several disorders including rheumatoid arthritis, cancers, pulmonary tuberculosis, etc. [21,22,23,24,25,26,27], and also in many skin disorders such as atopic dermatitis, hidradenitis suppurativa, leishmaniasis, and systematic lupus erythematosus [28,29,30]. This evidence concerns the gene IL32 and pulmonary tuberculosis.