Selected ILC populations have been reported to contribute to the pathogenesis of inflammatory skin diseases, such as ILC2 in atopic dermatitis and ILC3 in psoriasis (Batista et al., 2013, Imai et al., 2013, Kim et al., 2013, Pantelyushin et al., 2012, Salimi et al., 2013, Teunissen et al., 2014, Villanova et al., 2014). The gene discussed is CCL27; the disease is psoriasis.