To further confirm that TRPV2 activity is recruited during bacterial infection and contributes to phagocytosis, we investigated TRPV2 subcellular localization in response to P. aeruginosa. Confocal pictures analysis of cells infected with P. aeruginosa for 5, 10, 30 or 60 min showed an increase of TRPV2 level at the plasma membrane from 30 min, and a reduction at 60 min. The gene discussed is TRPV2; the disease is bacterial infectious disease.