27 found that ENO1 also played important roles in glioma, and Fu et al. 28 reported that ENO1 was overexpressed in nonsmall cell lung cancer (NSCLC) and promoted glycolysis, proliferation, migration, invasion, and tumorigenesis by activating the FAK‐mediated PI3K/AKT pathway. This evidence concerns the gene ENO1 and non-small cell lung carcinoma.