However, clinical outcomes were not significantly different among patients with long-chain FAODs, including VLCAD deficiency and LCHAD/MTP deficiencies identified either by newborn screening or by symptomatic presentation; most long-chain FAOD patients developed recurrent rhabdomyolysis and hypertrophic cardiomyopathy regardless of presymptomatic management. This evidence concerns the gene HADHA and rhabdomyolysis.