While the levels of MCP-1 were decreased in NKLAM-KO lungs at 24 and 48h, the levels of MIP1α at 48h were significantly increased compared to the 24h timepoint yet the levels were similar between genotypes (Fig 5) suggesting that other signaling mechanisms may come into play later in infection that normalize leukocyte migration in the NKLAM-KO mice. The gene discussed is CCL3; the disease is infection.