NFKB1 and neoplasm: Human hypopharyngeal primary cells exposed to acidic bile with BAY 11‐7082 demonstrated a significant decrease in “oncomirs” miR‐21 (P = .006), miR‐155 (P = .0035) and particularly of miR‐192 levels (P < .00001) (Figure 2B‐a), as well as a significant increase in “tumour suppressor” miR‐34a (P = .00023), miR‐375 (P = .00134) and particularly of miR‐451a levels (P < .000001) (Figure 2B‐b), compared to HHPC exposed to acidic bile without NF‐κB inhibitor (t test analysis; multiple comparisons by Holm‐Sidak).