As shown in Fig. 7a, overexpression of miR-1468 significantly inhibited, while miR-1468 knockdown promoted PPAR-γ/AKT pathway in HCC cells (P < 0.05, Fig. 7a), with change of p-AKT level rather than total AKT (P < 0.05, Fig. 7a). Here, AKT1 is linked to hepatocellular carcinoma.