Recently, Valera and colleagues generated a rodent oligodendroglial cell line, CG4, co‐infected with Lentivirus expressing human alpha‐synuclein or control and microRNA‐101 (miR‐101a‐3p) or control vector.61 The aim of the study was to investigate the potential pathogenic role of microRNA‐101 dysregulation in in vivo and in vitro models of MSA. The gene discussed is SNCA; the disease is multiple system atrophy.