KCNJ2 and catecholaminergic polymorphic ventricular tachycardia: KCNJ2 mutations responsible for the Andersen–Tawil syndrome typically manifest with the phenotypic triad of ventricular arrhythmias, commonly induced by exercise, periodic paralysis, and facial and limb dysmorphism, but these latter features might be variably present, which, together with a mild QTc prolongation, may very much mimic CPVT [159].