Having in mind that vimentin is a canonical marker of EMT, the overexpression of which is correlated with increased tumor growth, a lack of differentiation, invasion, the occurrence of metastases, and a poor prognosis in NSCLC and other tumors [52,53] it seems disturbing that fisetin, at physiologically attainable concentration, resulted in vimentin-rich network in lung cancer cells. This evidence concerns the gene VIM and non-small cell lung carcinoma.