Stimulation of the DK-MGhigh or primary tumor cells with EGF did not result in elevated phosphorylation of the mutant receptor, despite the marked increase in phosphorylation of the wild-type EGFR (Figure 1B–1F, Supplementary Figure 3), which is in contrast to previous reports [11, 14, 16, 29]. The gene discussed is EGFR; the disease is neoplasm.