The mutations or abnormal expression of these genes have been linked to aberrant molecular events leading to cancer, such as the interruption of p53 stabilization (MYH9) [57], abnormal chromatin/histone modification (KMT2D) [58], and aberrant Keap1-Nrf2 pathway (KEAP1) activity [55, 59]. This evidence concerns the gene MYH9 and cancer.