Several papers [30, 31] have reported an antitumor effect of PPARγ agonists (such as thiazolidinedione, prostaglandin, and their metabolic derivatives) in gastric cancer tissues, where PPARγ agonists disturb the growth cycle of tumor cells, induce tumor cell differentiation [28], promote tumor cell apoptosis, repress tumor cell proliferation and metastasis [32], and reduce tumor angiogenesis, and these effects are dose-dependent. The gene discussed is PPARG; the disease is gastric cancer.