The overexpression of CEACAM6, which was very likely from the myeloid cells (e.g., macrophages, monocytes, and granulocytes), suggests that apart from its well-known involvement in the anoikis resistance of many epithelial carcinomas (52), further investigations of the role it plays in tumor-associated immune cells hold promise. The gene discussed is CEACAM6; the disease is carcinoma.