Moreover, phosphorylation of p70-RSK was noted to be reduced in ABC-, but not in GC-DLBCLs, upon either C75 treatment or FASN depletion, indicating that active p70-RSK may be driving the USP11-eIF4B interaction for sustained oncogene expression (Supplementary Figure 33). This evidence concerns the gene EIF4B and aneurysmal bone cyst.