IFNG and chromomycosis: Accordingly, we hope to establish a chronic chromoblastomycosis model in mice by intraperitoneal injection with F. pedrosoi-spores, -hyphae and sclerotic cells respectively in the present study and aim to verify our hypothesis that the transformation of this etiological agent into sclerotic cell form can result in a compromised IFN-γ production via a mechanism involving chitin accumulation on the surface of sclerotic cells, and therefore promotes the refractoriness of experimental murine chromoblastomycosis.