The results indicated an overrepresentation of several immune response pathways such as “diabetes,” “autoimmune thyroid disease,” “IL22 signaling,” and “eicosanoid receptors,” particularly with the involvement of the HLA‐DOA, PTPRN2, HLA‐E, TPO, CEBPD, CFD, LTB4R and LTB4R2 genes in the childhood cALD samples. Here, PTPRN2 is linked to diabetes mellitus.