The results showed that si-CASC2 significantly up-regulated tumor cell viability, while miR-24 inhibition and miR-221 inhibition significantly down-regulated tumor cell viability; the promotive effect of si-CASC2 on tumor cell viability could be partially reversed by miR-24 inhibition or miR-221 inhibition (Figs. 6a,b). Here, CASC2 is linked to neoplasm.