Microbes induce epithelial-to-mesenchymal transitions (EMT) through various signaling pathways, such as TGFβ, Wnt, and Notch, which work together with the transcription factors (TFs) Slug, SNAIL, Twist, ZEB1, and ZEB2 to suppress E-cadherin, leading to tumor invasion, metastasis, and acquired drug resistance [135], [136], [137]. This evidence concerns the gene CDH1 and neoplasm.