VSV induces faster apoptosis in transformed cells having mutations in myelocytomatosis (Myc), Rat sarcoma (Ras), or tumor protein 53 (p53) genes, as well as tumors overexpressing Bcl-2 genes that are associated with uncontrolled cell division, metastasis, and angiogenesis [45]. The gene discussed is TP53; the disease is sarcoma.