Recent pharmacological studies found that Rg1 is able to suppress myocardial infarction area (Wang et al., 2010), enhance the myocardial perfusion and preserve left ventricle (LV) function (Wei et al., 2007; Yin et al., 2011), as well as ameliorate ventricular remodeling in acute or chronic myocardial infarction animal model (Yin et al., 2011). The gene discussed is PPP1R3A; the disease is myocardial infarction.